Ex Parte Sander-Struckmeier et al - Page 3




             Appeal No. 2005-1150                                                                              
             Application No. 09/953,450                                                                        
                   The references relied upon by the examiner are:                                             
             Fallis et al. (Fallis), “Observations on some metabolic changes after total                       
             pancreatoduodenectomy,” Annals of Surgery, pgs. 639-667, 1948.                                    
             Delhaye et al. (Delhaye), “Comparative evaluation of a high lipase pancreatic enzyme              
             preparation and a standard pancreatic supplement for treating exocrine pancreatic                 
             insufficiency in chronic pancreatitis,” European Journal of Gastroenterology and                  
             Hepatology, vol. 8, no. 7, pgs. 699-703, 1996.                                                    
                   Claims 1-7 stand rejected under 35 U.S.C. § 103(a) as being unpatentable over               
             Fallis in view of Delhaye.                                                                        
                   We reverse, and set forth a new ground of rejection under 37 C.F.R. § 41.50(b)              
             with respect to claims 1 and 3-7.  Absent further action by the examiner, claim 2 stands          
             free of the prior art.                                                                            
                                                 Background                                                    
                   As indicated by the claims above, the present invention is directed to a method of          
             treating primary diabetes mellitus Type I.  The specification discloses that Type I               
             diabetes is due to an insulin deficiency (pages 1 and 7); and Type II diabetes is due to          
             reduced insulin effectiveness (page 1).  Thus, the specification defines Type I diabetes          
             as being insulin dependent; whereas Type II is non-insulin-dependent.  Id.  These                 










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